A slowly developing and persistent autoimmune disease caused by the action of certain antibodies that attack the tissues of the thyroid gland; also known as chronic lymphocytic or autoimmune thyroiditis. Hashimoto thyroiditis can eventually cause hypothyroidism (underactivity of the thyroid gland). In Hashimoto thyroiditis, thyroid tissue is invaded by white blood cells called lymphocytes. The invasion produces inflammation, degeneration, and scarring of the tissue within the thyroid gland, gradually decreasing the gland’s ability to produce and release hormones.
An autoimmune illness in which there is inflammation, and then destruction and fibrosis of the thyroid gland, ultimately resulting in hypothyroidism. Autoantibodies against thyroglobulin and receptors for thyroid stimulating hormone cause the progressive destruction. Hashimoto’s is the most common cause of hypothyroidism in areas of the world where there are inadequate levels of iodine. It affects people of all ages, but is most common in older women and those with a family history of autoimmune diseases. Thyroid hormone replacement is required.
Hashimoto’s thyroiditis is an autoimmune disorder where the body’s immune system generates antibodies that attack the thyroid gland cells. Consequently, the thyroid gland cannot produce enough thyroid hormones, leading to a condition known as hypothyroidism. This condition is eight times more prevalent in women than in men. The main symptoms of Hashimoto’s thyroiditis include tiredness, muscle weakness, weight gain, and an enlarged thyroid gland.
A positive diagnosis is established through blood tests that reveal the presence of antithyroid antibodies and low levels of thyroid hormones. The recommended treatment involves lifelong thyroid hormone replacement therapy. Additionally, in certain cases, individuals may develop other autoimmune disorders alongside Hashimoto’s thyroiditis.