Category: P

  • Physiotype

    A population of a pathogen in which all individuals have a particular character of physiology (but not of pathogenicity) in common’ (Robinson, 1969); physiodeme, which would appear to be the preferred usage.  

  • Phage resistant

    Typically, the resistance to infection occurs as a result of the phage’s inability to adhere to the bacterial cell, thus failing to cause any harm.    

  • Perthophyte

    A living entity that sustains itself by consuming deceased organic matter present within the body of a living host. This can be compared with a saprophyte, which also feeds on dead organic matter.  

  • Permitted tolerance

    The uppermost degree of toxic substances admitted (e.g., lead or arsenic) in comestibles designed for human ingestion.  

  • Pelleting

    Coating of seed with inert material, often incorporating pesticides, to ensure uniform size and shape.  

  • Vertical pathogenicity

    Pathogenicity refers to the ability of a pathogen to cause disease in a host. This ability may be effective against certain varieties of the host but not against others. Furthermore, pathogenicity can be expressed in a manner that is different when interacting with various races and varieties. It is noteworthy that the use of this…

  • Prayer therapy

    The utilization of supplication to an elevated force to aid in the fight against ailment and the reestablishment of wellbeing.  

  • Phytotherapeutics

    The overarching utilization of botanical constituents in the management of ailments and preservation of well-being.  

  • Placebo response

    A therapeutic effect achieved by the administration of a fake treatment. In the case of pain, placebo responses are reductions of pain sensation caused by proce¬ dures that don’t contain active principles. Placebo responses can be achieved by suggestion, expectation, or conditioning.  

  • Pattern theory of pain

    A theoretical interpretation that proposes that pain is a result of the pattern of activation of peripheral sensors and central-nervous-system neurons and not of a chain of specific pain-related neurons. The emphasis of the pattern theory is on how brain neurons are activated by pain-producing stimuli rather than on what specific neurons are activated.